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The proposed mechanisms include promotion of angiogenesis (new blood-vessel growth into damaged tissue), modulation of growth-factor pathways including VEGF and basic FGF, and influence on the nitric-oxide signalling system that governs how blood vessels respond to injury

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And so my thinking in reading the preclinical literature, the animal literature, was that, all right, that makes a lot of sense in the context of those species, but we've never seen any evidence in our trial

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