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Moreover, forced expression of YAP was able to prevent cell death induced by sorafenib, suggesting that YAP/TAZ act as inhibitors of ferroptosis in HCC cells ( It was postulated that YAP/TAZ target the SLC7A11 to affect ferroptosis in HCC cells, thus contributing to resistance to sorafenib ( Activating transcription factor 4 (ATF4) is a critical mediator of oxidative homeostasis and has been validated to inhibit ferroptosis induced by sorafenib, erastin, and RSL3 via acting on SLC7A11 ( In short, YAP/TAZ are negative regulators of ferroptosis in HCC cells through activating SLC7A11 dependent on the TEAD motif, and through interacting with ATF4 which is a promotor of SLC7A11 expression