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These molecules can bind to pattern recognition receptors, including Toll-like receptors (TLRs), receptors for advanced glycation end products (RAGEs), and purinergic receptors on liver immune cells, resulting in the activation of immune cells
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reported that up-regulated β-catenin was involved in the initiation and development of papilloma induced by DMBA/TPA by maintaining the activity of CD34-positive HFSCs [19]
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