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For instance, in male C57BL/6 mice, PEA treatment significantly ameliorated mechanical hyperalgesia and inflammation in a chronic constriction injury model and was also shown to be neuroprotective, preserving nerve structure, all absent in PPAR-α knockout mice, confirming the PPAR-α signal-dependent analgesic and neuroprotective actions of PEA (Di Cesare Mannelli et al., 2013)

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